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Anti-stress
"Descrizione"
by Al222 (19785 pt)
2023-Dec-30 18:23

Components that help in anti-stress action are substances that can contribute to reducing stress and promoting relaxation. Here are some examples:

  • Magnesium. Helps relax muscles and calm the nervous system.
  • Vitamin B. B-complex vitamins support the nervous system and reduce fatigue.
  • Omega-3 Fatty Acids. Reduce inflammation and can help improve mood.
  • Green Tea. Contains L-theanine, which can promote relaxation without drowsiness.
  • Ashwagandha. An adaptogenic herb that can help reduce stress and anxiety.
  • Chamomile. Known for its calming and relaxing properties.
  • Melatonin. Useful for regulating sleep, important for stress management.
  • Rhodiola Rosea. Another adaptogenic herb that can help combat fatigue and stress.
  • Ginseng. Can improve resistance to stress and increase energy.
  • Regular Physical Exercise. Helps reduce stress and improve overall well-being.

Components that can contribute to or exacerbate stress include various factors that can increase stress levels or worsen its effects. Here are some examples

  • Caffeine. Found in coffee, tea, and some sodas, it can increase anxiety and irritability.
  • Refined Sugars. Present in sweets and snacks, can cause blood sugar fluctuations and increase stress.


  • Alcohol. May seem relaxing, but actually increases stress levels in the body.
  • Processed Foods. Often high in fats and sugars, can negatively affect mood and mental health.
  • Nicotine. Found in tobacco products, can increase tension and stress.
  • Lack of Sleep. Sleep deprivation can significantly increase stress levels.
  • Work Overload. Excessive workload can be a significant source of stress.
  • Interpersonal Conflicts. Problems in relationships can increase stress.
  • Noise and Environmental Pollution. Can increase the feeling of stress.
  • Lack of Physical Activity. Exercise helps reduce stress, so its absence can contribute to increasing it.

The reports  provided on Tiiips website are for informational purposes only and should not replace medical advice. Always consult a healthcare professional before making health-related decisions.

References_____________________________________________________________________

Dragoş D, Tănăsescu MD. The effect of stress on the defense systems. J Med Life. 2010 Jan-Mar;3(1):10-8. PMID: 20302192; PMCID: PMC3019042.

Abstract. Acute stress increases resistance to infection. The alteration of this mechanism in chronically stressed people impairs the organism's ability to mount a strong immune response with a resultant increase in morbidity. Acute stress induces a probable sympatho-adrenergically mediated increase in chemotaxis and adhesion molecules expression, thus promoting immune cells migration to sites of infection and/or inflammation, while chronic stress impairs this mechanism. Protracted stressful conditions decrease NK cytotoxic capacity. There is a substance P, which under stressful circumstances mediates the increase in macrophage cytokine production. Acute stress increases T cell mobilization through a beta2-adrenergically mediated process, which is blunted during chronic stress. Psychological stress impairs the immune system's ability to produce antibodies in response to a vaccine, thereby making the organism more vulnerable to infections.

Rubartelli A, Sitia R. Stress as an intercellular signal: the emergence of stress-associated molecular patterns (SAMP). Antioxid Redox Signal. 2009 Oct;11(10):2621-9. doi: 10.1089/ars.2009.2377. 

Abstract. Cells are continuously exposed to stressful situations that generally entail generation of reactive oxygen species and other redox alterations. Low levels of stress are physiological and can transmit essential survival or adaptive signals. At higher levels, however, the responses become maladaptive and cause damage. Frequently, stressful events occurring in a few cells propagate, resulting in tissue or even systemic response. Here we review recent evidence suggesting that stressed cells signal their state by expressing on their surface and secreting suitable molecular clues, which we propose to term Stress-Associated Molecular Patterns (SAMP). A unifying mechanism seems to involve the release of oxidoreductases and redox modifiers into the intercellular space, with structural and functional alterations in key signaling molecules. These observations open the way to novel therapeutic strategies.

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